Translesion synthesis by Y family DNA polymerases bypasses lesions on DNA template

Recognition of DNA damage by PCNA-containing replication complex

Recognition and association of DNA glycosylase with site containing an affected pyrimidine

Recognition and association of DNA glycosylase with site containing an affected purine

POLB-Dependent Long Patch Base Excision Repair

Resolution of AP sites via the multiple-nucleotide patch replacement pathway

Resolution of AP sites via the single-nucleotide replacement pathway

SLBP independent Processing of Histone Pre-mRNAs

Activation of BIM and translocation to mitochondria

Activation of BAD and translocation to mitochondria

Activation of NOXA and translocation to mitochondria

Activation and oligomerization of BAK protein

BH3-only proteins associate with and inactivate anti-apoptotic BCL-2 members

Release of apoptotic factors from the mitochondria

Activation of caspases through apoptosome-mediated cleavage

SMAC(DIABLO)-mediated dissociation of IAP:caspase complexes

CaMK IV-mediated phosphorylation of CREB

ALKBH3 mediated reversal of alkylation damage

Electric Transmission Across Gap Junctions

Presynaptic depolarization and calcium channel opening

Neurotransmitter uptake and metabolism In glial cells

Neurotransmitter receptors and postsynaptic signal transmission

Formation of RNA Pol II elongation complex

Formation of the Early Elongation Complex

Inhibition of replication initiation of damaged DNA by RB1/E2F1

E2F-enabled inhibition of pre-replication complex formation

E2F mediated regulation of DNA replication

Activation, translocation and oligomerization of BAX

Downstream signaling events of B Cell Receptor (BCR)

Antiviral mechanism by IFN-stimulated genes

Tolerance of reactive oxygen produced by macrophages

Latent infection - Other responses of Mtb to phagocytosis

Tolerance by Mtb to nitric oxide produced by macrophages

Regulation of gene expression by Hypoxia-inducible Factor

Oxygen-dependent proline hydroxylation of Hypoxia-inducible Factor Alpha

Constitutive Signaling by Ligand-Responsive EGFR Cancer Variants