Endosomal Sorting Complex Required For Transport (ESCRT)

Negative regulators of RIG-I/MDA5 signaling

IKK complex recruitment mediated by RIP1

Class I peroxisomal membrane protein import

Insertion of tail-anchored proteins into the endoplasmic reticulum membrane

Assembly and cell surface presentation of NMDA receptors

FOXO-mediated transcription of oxidative stress, metabolic and neuronal genes

Postmitotic nuclear pore complex (NPC) reformation

Negative regulation of NMDA receptor-mediated neuronal transmission

EGR2 and SOX10-mediated initiation of Schwann cell myelination

Response of EIF2AK4 (GCN2) to amino acid deficiency

Estrogen-dependent nuclear events downstream of ESR-membrane signaling

EML4 and NUDC in mitotic spindle formation

ADORA2B mediated anti-inflammatory cytokines production

Purinergic signaling in leishmaniasis infection

Sensory processing of sound by inner hair cells of the cochlea

Sensory processing of sound by outer hair cells of the cochlea

Anti-inflammatory response favouring Leishmania parasite infection

Sealing of the nuclear envelope (NE) by ESCRT-III

Inhibition of DNA recombination at telomere

Signaling by phosphorylated juxtamembrane, extracellular and kinase domain KIT mutants

Diseases of DNA Double-Strand Break Repair

Signaling by CSF1 (M-CSF) in myeloid cells

Aberrant regulation of mitotic cell cycle due to RB1 defects

SARS-CoV-1 activates/modulates innate immune responses

Defective homologous recombination repair (HRR) due to BRCA2 loss of function

Defective homologous recombination repair (HRR) due to BRCA1 loss of function

Defective homologous recombination repair (HRR) due to PALB2 loss of function

Defective HDR through Homologous Recombination Repair (HRR) due to PALB2 loss of BRCA1 binding function

Defective HDR through Homologous Recombination Repair (HRR) due to PALB2 loss of BRCA2/RAD51/RAD51C binding function

SARS-CoV-2 activates/modulates innate and adaptive immune responses

Sensory perception of sweet, bitter, and umami (glutamate) taste

Signaling by ALK fusions and activated point mutants